Product Information
RGAM005 targets Mouse IgG (H+L) in FC and IF. It can bind to all mouse IgG isotypes, mouse IgM and IgA as well as rat IgG. It shows minimal reactivity for rabbit, human, pig and bovine IgGs.
| Concentration | 0.5 mg/mL |
| Applications | IF, FC |
| Host Species | Goat |
| Species Reactivity | Mouse |
| Physical State | Liquid |
| Suggested Dilution Range | 1:200-1:1000 for IF and FC |
| Purity | The antibody was purified from culture media supernatant by immunoaffinity chromatography using Protein G beads. |
| Storage Buffer | PBS with 50% glycerol, 10 mg/mL BSA, 0.1% Proclin-300, pH 7.4. |
| Storage Instructions | Store at -20°C. Stable for one year after shipment. |
| Fluorophore | CoraLite® Plus 647, Amax=654 nm, Emax=674 nm |
| Clonality | Multiclonal recombinant |
| RRID | AB_3073503 |
| Note | This product is for research use only,not for diagnostic or therapeutic use. |
Background
Multi-rAb™ Recombinant Secondary Antibodies are mixtures of precisely engineered recombinant monoclonal antibodies that recognize multiple complementary epitopes on the same IgG. Each recombinant clone in the final multiclonal mixture is carefully selected after rigorous characterization and screening to ensure the highest level of performance. The selected clones are mixed in various combinations, each of which is then functionally characterized in multiple applications to select the final multiclonal mixture with optimal performance.
Each lot of Multi-rAb™ Secondary Antibodies is produced by mixing the same high performing recombinant clones every time to ensure that they have high batch-to-batch consistency, which in turn results in high reproducibility of experimental data.
Publications
| Title |
|---|
Nat Commun Multi-protomics analysis identified host cellular pathways perturbed by tick-borne encephalitis virus infection |
ACS Nano Trojan Horse Strategy for Wireless Electrical Stimulation-Induced Zn2+ Release to Regulate Neural Stem Cell Differentiation for Spinal Cord Injury Repair |
Exp Mol Med Cholesterol biosynthesis induced by radiotherapy inhibits cGAS-STING activation and contributes to colorectal cancer treatment resistance |
